wael_gamaleldin

wael_gamaleldin

Assistant Professor - professor

Faculty of medicine

Address: sohag university hospital - urology dept

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Publications Which contain the keyword: Psoriasis Vulgaris


2023-02-13 14:27:30 Keywords Psoriasis Vulgaris, Psoriasis,
Assessment of LC3 autophagy marker in psoriasis vulgaris patients with metabolic syndrome: a cross-sectional study
Purpose: Psoriasis vulgaris, one of the most prevalent chronic inflammatory skin diseases, is associated with metabolic syndrome (MetS). Autophagy, an intracellular degradation system is essential for cell survival and differentiation, and its dysfunction may contribute to metabolic diseases. A cross-sectional study was conducted on 38 psoriasis vulgaris patients and 16 healthy control subjects to 1) Assess immunohistochemical (IHC) expression of ... Read more

2020-12-22 22:58:31 Keywords Psoriasis Vulgaris,
Assessment of the Effect of Metabolic Syndrome on the Autophagy Marker LC3 in Psoriasis Vulgaris Patients: A Cross-Sectional Study.
Purpose: Psoriasis vulgaris, one of the most prevalent chronic inflammatory skin diseases, is associated with metabolic syndrome (MetS). Autophagy, an intracellular degradation system is essential for cell survival and differentiation, and its dysfunction may contribute to metabolic diseases. A cross-sectional study was conducted on 38 psoriasis vulgaris patients and 16 healthy control subjects to 1) Assess immunohistochemical (IHC) expression of ... Read more

2018-10-22 23:23:31 Keywords Psoriasis Vulgaris, IL-6, Bcl-2, Serum Soluble Fas,
Serum Soluble Fas, Bcl-2 Proteins and IL-6 Levels in Patients with Psoriasis Vulgaris and their correlations to disease activity.
INTRODUCTIONF: as (Apo-I /CD95) is a widely expressed membrane-anchored protein that induces apoptosis. Keratinocytes constitutively express the Fas antigen and Fas- mediated apoptosis may characterize several pathological conditions affecting human skin. Soluble Fas (sFas) has been demonstrated in serum by alternative mRNA splicing of Fas receptor. It can antagonize cell surface Fas function by downregulating Fas- mediated apoptosis and its ... Read more

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