Background: Female Pattern Hair Loss (FPHL), also known as female androgenetic alopecia (AGA), is the leading cause of hair loss in adult women, with a significant impact on patients' quality of life. It develops from progressive follicle thinning, which leads to a decrease in hair density, resulting in non-scarring widespread alopecia with distinct clinical, dermoscopic, and histological patterns. The lack of knowledge about the etiology of the hair-thinning process and the factors regulating follicular development limits the promise of novel therapeutics. Topical minoxidil, used to treat androgenetic alopecia since the 1990s, is the most highly evidenced medication and remains the first choice. However, some patients do not show improvement with it, so it is crucial to seek alternative lines of treatment. It is commonly recognized that maintaining the regular hair cycle requires keratinocytes to express vitamin D receptors. The writers of this article examine vitamin D's potential contribution to hair development in a critical manner. Objectives: This paper will provide a brief overview of vitamin D biology within the hair follicle, its role in the etiology of hair loss, and the justification for supplementing in FPHL. Conclusion: A number of signaling pathways that control the proliferation and differentiation of hair follicles depend critically on vitamin D. Many studies reveal a link between serum vitamin D levels and FPHL, as well as other types of alopecia. Nonetheless, there aren't enough compelling studies to back up the utility of vitamin D analogues in treating these conditions and reversing hair loss. As a result, further research is required before vitamin D is consistently proposed as a therapy option for these illnesses.